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J Fombonne

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J
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Fombonne
Robinson, R. A., Lange, M. B., Sultana, R., Fombonne, J., Gorostiza, O., Zhang, J., et al. (2011). Differential expression and redox proteomics analyses of an Alzheimer disease transgenic mouse model: effects of the amyloid-β peptide of amyloid precursor protein. Neuroscience, 177, 207-22. https://doi.org/10.1016/j.neuroscience.2011.01.005 (Original work published 2011)
Sultana, R., Robinson, R. A., Lange, M. B., Fiorini, A., Fombonne, J., Baker, A., et al. (2012). Do proteomics analyses provide insights into reduced oxidative stress in the brain of an Alzheimer disease transgenic mouse model with an M631L amyloid precursor protein substitution and thereby the importance of amyloid-beta-resident methionine 35 in.. Antioxidants & Redox Signaling, 17(11), 1507-14. https://doi.org/10.1089/ars.2011.4470 (Original work published 2012)
Butterfield, D. A., Lange, M. B., Tang, H., Sowell, R. A., Spilman, P., Fombonne, J., et al. (2010). In vivo oxidative stress in brain of Alzheimer disease transgenic mice: Requirement for methionine 35 in amyloid beta-peptide of APP. Free Radical Biology & Medicine, 48(1), 136-44. https://doi.org/10.1016/j.freeradbiomed.2009.10.035 (Original work published 2010)