Skip to main content

E Barone

First name:
E
Last name:
Barone
Perluigi, M., Barone, E., Di Domenico, F., & Butterfield, D. A. (2016). Aberrant protein phosphorylation in Alzheimer disease brain disturbs pro-survival and cell death pathways. Biochimica Et Biophysica Acta, 1862(10), 1871-82. https://doi.org/10.1016/j.bbadis.2016.07.005
Tramutola, A., Di Domenico, F., Barone, E., Arena, A., Giorgi, A., di Francesco, L., et al. (2017). Polyubiquitinylation Profile in Down Syndrome Brain Before and After the Development of Alzheimer Neuropathology. Antioxidants & Redox Signaling, 26(7), 280-298. https://doi.org/10.1089/ars.2016.6686 (Original work published 2017)
Tramutola, A., Arena, A., Cini, C., Butterfield, D. A., & Barone, E. (2017). Modulation of GLP-1 signaling as a novel therapeutic approach in the treatment of Alzheimer's disease pathology. Expert Review Of Neurotherapeutics, 17(1), 59-75. https://doi.org/10.1080/14737175.2017.1246183
Di Domenico, F., Barone, E., Perluigi, M., & Butterfield, D. A. (2017). The Triangle of Death in Alzheimer's Disease Brain: The Aberrant Cross-Talk Among Energy Metabolism, Mammalian Target of Rapamycin Signaling, and Protein Homeostasis Revealed by Redox Proteomics. Antioxidants & Redox Signaling, 26(8), 364-387. https://doi.org/10.1089/ars.2016.6759 (Original work published 2017)
Barone, E., Di Domenico, F., Cassano, T., Arena, A., Tramutola, A., Lavecchia, M. A., et al. (2016). Impairment of biliverdin reductase-A promotes brain insulin resistance in Alzheimer disease: A new paradigm. Free Radical Biology & Medicine, 91, 127-42. https://doi.org/10.1016/j.freeradbiomed.2015.12.012
Tramutola, A., Di Domenico, F., Barone, E., Perluigi, M., & Butterfield, D. A. It Is All about (U)biquitin: Role of Altered Ubiquitin-Proteasome System and UCHL1 in Alzheimer Disease. Oxidative Medicine And Cellular Longevity, 2016, 2756068. https://doi.org/10.1155/2016/2756068 (Original work published 2016)
Tramutola, A., Pupo, G., Di Domenico, F., Barone, E., Arena, A., Lanzillotta, C., et al. Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype. Journal Of Alzheimer's Disease : Jad, 52(1), 359-371. https://doi.org/10.3233/JAD-151105 (Original work published 2016)
Barone, E., Mancuso, C., Di Domenico, F., Sultana, R., Murphy, M. P., Head, E., & Butterfield, D. A. (2012). Biliverdin reductase-A: a novel drug target for atorvastatin in a dog pre-clinical model of Alzheimer disease. Journal Of Neurochemistry, 120(1), 135-46. https://doi.org/10.1111/j.1471-4159.2011.07538.x
Barone, E., Cenini, G., Sultana, R., Di Domenico, F., Fiorini, A., Perluigi, M., et al. (2012). Lack of p53 decreases basal oxidative stress levels in the brain through upregulation of thioredoxin-1, biliverdin reductase-A, manganese superoxide dismutase, and nuclear factor kappa-B. Antioxidants & Redox Signaling, 16(12), 1407-20. https://doi.org/10.1089/ars.2011.4124 (Original work published 2012)
Cenini, G., Dowling, A. L., Beckett, T. L., Barone, E., Mancuso, C., Murphy, M. P., et al. (2012). Association between frontal cortex oxidative damage and beta-amyloid as a function of age in Down syndrome. Biochimica Et Biophysica Acta, 1822(2), 130-8. https://doi.org/10.1016/j.bbadis.2011.10.001